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HCEMM-SU Molecular Channelopathies Research Group

Publications

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6. Blue flash sheds light on the roles of individual phosphoserines in CFTR channel activation.

Csanády L.
Journal/Proc./Book: Journal of General Physiology
Year: 2023
Thematic Pillar: Metabolic and Cardiovascular Diseases
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5. Optimization of CFTR gating through the evolution of its extracellular loops.

Simon MA, Csanády L.
Journal/Proc./Book: Journal of General Physiology
Year:  2023
Thematic Pillar: Metabolic and Cardiovascular Diseases

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4. Dual amplification strategy turns TRPM2 channels into supersensitive central heat detectors.

Bartók Á, Csanády L.
Journal/Proc./Book: Proceedings of the National Academy of Sciences of the United States of America
Year: 2022
Thematic Pillar: Metabolic and Cardiovascular Diseases
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3. Molecular pathology of the R117H cystic fibrosis mutation is explained by loss of a hydrogen bond.

Simon MA, Csanády L.
Journal/Proc./Book: eLife Sciences Publications
Year: 2021
Thematic Pillar: Metabolic and Cardiovascular Diseases

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2. Degenerate but indispensable: how CFTR channel activity depends on the catalytically inactive ATP binding site?

László Csanády
Journal/Proc./Book: The Journal of Physiology
Year: 2021
Thematic Pillar: Metabolic and Cardiovascular Diseases

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1. Simple binding of protein kinase A prior to phosphorylation allows CFTR anion channels to be opened by nucleotides

Csaba Mihályi, Iordan Iordanov, Beáta Töröcsik, László Csanády
Journal/Proc./Book: Proceedings of the National Academy of Sciences
Year: 2020
Thematic Pillar: Metabolic and Cardiovascular Diseases

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This project has received funding from the European Union’s Horizon 2020 research and innovation programme under grant agreement No 739593. HCEMM supported by EU Programme: H2020-EU.4.a. – Teaming of excellent research institutions and low performing RDI regions. Project starting date was 1 April 2017.

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