HCEMM related publications

HCEMM-USZ Translational Pancreatology Research Group

Publications

4. Dual amplification strategy turns TRPM2 channels into supersensitive central heat detectors.

Bartók Á, Csanády L.
Journal/Proc./Book: Proceedings of the National Academy of Sciences of the United States of America
Year: 2022
Thematic Pillar: Metabolic and Cardiovascular Diseases

3. Molecular pathology of the R117H cystic fibrosis mutation is explained by loss of a hydrogen bond.

Simon MA, Csanády L.
Journal/Proc./Book: eLife Sciences Publications
Year: 2021
Thematic Pillar: Metabolic and Cardiovascular Diseases

2. Degenerate but indispensable: how CFTR channel activity depends on the catalytically inactive ATP binding site?

László Csanády
Journal/Proc./Book: The Journal of Physiology
Year: 2021
Thematic Pillar: Metabolic and Cardiovascular Diseases

1. Simple binding of protein kinase A prior to phosphorylation allows CFTR anion channels to be opened by nucleotides

Csaba Mihályi, Iordan Iordanov, Beáta Töröcsik, László Csanády
Journal/Proc./Book: Proceedings of the National Academy of Sciences
Year: 2020
Thematic Pillar: Metabolic and Cardiovascular Diseases

HCEMM-USZ Translational Pancreatology Research Group

Publications

4. Dual amplification strategy turns TRPM2 channels into supersensitive central heat detectors.

3. Molecular pathology of the R117H cystic fibrosis mutation is explained by loss of a hydrogen bond.

2. Degenerate but indispensable: how CFTR channel activity depends on the catalytically inactive ATP binding site?

1. Simple binding of protein kinase A prior to phosphorylation allows CFTR anion channels to be opened by nucleotides

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